It has been suggested by conventional practitioners that testosterone is the fuel that ignites the growth of the prostate and prostate cancer. However, the question arises why younger men with high testosterone levels are not likely to have benign prostate or prostate cancer and older men with low testosterone develop benign or prostate cancer. When testosterone levels become low in older men, their estrogen level rises and their DHEA is depleted. This would suggest that it is not the testosterone itself that causes the problem. It is most likely contributed to the low level of testosterone, their high estrogen level, and the reduction of other essential hormones. If testosterone were the cause of prostate cancer, young men would be dying from prostate cancer. Many studies have shown that men with the highest level of testosterone have the least prostate enlargement and men with the highest level of estrogen have enlarged prostates.
THE ABILITY OF HORMONES TO PREVENT & REVERSE PROSTATE CANCER
The ability of progesterone to prevent and reverse prostate cancer has been highly reported by Dr. John Lee and many well-known physicians and natural health practitioners for many years.
It is diﬃcult to understand why the medical establishment places the blame for prostate problems on testosterone and why they are not supportive of progesterone treatment. Natural progesterone should not be mistaken for synthetic progestins. They are molecularly diﬀerent, and natural progesterone does not carry the same side eﬀects or risks that the synthetic one does.
Men produce estrogen (Estradiol), but in lower amounts than women. Many scientists and researchers have suggested the importance of the testosterone and estrogen ratio to normal prostate or diseased prostate. Men also produce progesterone, but only about half the amount produced by females. Progesterone is normally produced in men by the adrenal glands and testes. Unfortunately, male progesterone levels drop with aging, just as male testosterone does. It is suggested by scientists that severe, prolonged stress could also deplete progesterone. Progesterone is vital to good health in humans. It is suggested by scientists that it is the primary precursor of our adrenal cortical hormones and testosterone. The male hormone testosterone is antagonist to estradiol. Testosterone prevents estradiol from causing prostate cancer by destroying the prostate cancer cells it stimulates. Research studies have also shown that when prostate cells are exposed to estrogen, the cells proliferate and become cancerous. When progesterone or testosterone was added, cancer cells died.
DHEA is the most abundant steroid in the body, and it declines with age. Abnormally low levels have been reported to be related to a number of degenerative diseases, including cancer. Supplemental DHEA has been cited in several reports to have anticancer activity. DHEA, melatonin, and pregenolone levels fall very low in men after age forty. LH (luteinizing hormone) and FSH (follicle-stimulating hormone) rise in men as they age, which could cause problems in men’s health. It is recommended that if your DHEA is low you should supplement it with twenty-ﬁve mg of DHEA, and you should monitor your level every three to six months. healthy and nutrition food
Researchers suggest that there is increasing evidence that nutrition may play a signiﬁcant role in the prevention and/or progression of prostate cancer. There are data to suggest that diet may be of more signiﬁcance in the behaviour of prostate cancer than in either breast or colon cancer. Autopsy studies have demonstrated that there is evidence of microscopic prostate cancer cells in approximately 80 percent of men over eighty years old.
This ﬁnding is the same throughout the world and is unrelated to race. However, the incidence of clinically signiﬁcant prostate cancer is signiﬁcantly greater in Western countries as opposed to Oriental countries. In a study published in the British Journal of Cancer in 1991 by Dr. Schimizu and Associates, it was noted that within one generation, there was a fourfold to ninefold increase in prostate cancer among men who immigrated to the United States in contrast with their counterparts who remained in Japan.
In a similar study published in Acta Oncologica in 1991 by Dr. Muir and Associates, there was found a threefold to sevenfold increase in the incidence of prostate cancer in Oriental men who immigrated to the San Francisco Bay area. This suggests the presence of either an environmental or nutritional factor that may be playing a role in stimulating the growth of microscopic cancer cells to clinically signiﬁcant cancer in the United States, Canada, and some European countries.
Abstract from the Journal of Nutrition suggested that; “Because of their safety and the fact that they are not perceived as “medicine,” food-derived products are highly interesting for development as chemopreventive agents that may ﬁnd widespread, long-term use in populations at normal risk. Numerous diet-derived agents are included among the 40 promising agents and agent combinations that are being evaluated clinically as chemopreventive agents for major cancer targets.
These targets include breast, prostate, colon and lung. Examples include green and black tea polyphenols, soy isoﬂavones, Bowman- Birk soy protease inhibitor, curcumin, phenethyl isothiocyanate, sulforaphane, lycopene, indole-3-carbinol, perillyl alcohol, vitamin D, vitamin E, selenium and calcium. Many food-derived agents are extracts, containing multiple compounds or classes of compounds. For developing such agents, the National Cancer Institute (NCI) has advocated codevelopment of a single or a few putative active compounds that are contained in the food-derived agent. The active compounds provide mechanistic and pharmacologic data that may be used to characterize the chemopreventive potential of the extract, and these compounds may ﬁnd use as chemopreventives in higher risk subjects (patients with precancers or previous cancers). Other critical aspects to developing the food-derived products are careful analysis and deﬁnition of the extract to ensure reproducibility (e.g., growth conditions, chromatographic characteristics or composition), and basic science studies to conﬁrm epidemiologic ﬁndings associating the food product with cancer prevention.”
Of all these potential factors related to prostate cancer, the most signiﬁcant has been the relationship between dietary fat and prostate cancer. Among animal fat, fat from red meat was most strongly linked to advanced cancer. Those who consumed the most red meat were said to be two-and-a-half times more likely to have advanced cancer or to die from prostate cancer, than those who ate meat infrequently. The investigators of this report found a tenfold diﬀerence intake of animal fat between low and high consumers, ranging from an average of 3.2 grams of fat from red meat each day to 30.5 grams per day.
Fat intake was also associated with the risk of advanced cancer. Those who consumed an average of 88.6 grams of fat per day had a 76 percent greater chance of developing advanced prostate cancer, than those who averaged 53.2 grams of fat daily. There have been several well-executed studies involving more than 3,000 patients with prostate cancer and more than 4,600 patients without prostate cancer. Eleven of these studies have shown a positive association between increased dietary fat and a higher risk of prostate cancer, with some studies showing a greater than threefold increase in cancer incidence. In another group of studies of close to 100,000 men, there was also a positive association between dietary fat and the development of prostate cancer.
In a recent study published in the Journal of the National Cancer Institute in 1993 by Dr. Giovannucci and Associates, there was a marked increase in the likelihood of prostate cancer in men with a high dietary fat intake from red meat. These investigators found a strong relationship between the intake of linolenic acid, a fatty acid primarily associated with red meats, and prostate cancer. But Dr Giovannucci said most of this fatty acid was derived from animal fats rather than vegetable oils in men’s diets. He also suggested that there is a need for more research. When assessing this information, we must take into account, the fact that there are good fats and there are bad fats. There are essential fats the human body needs for survival…
There is a considerable amount of research available proving the existence of links between speciﬁc types of food and cancer. These links exist both for cancer in general and for prostatic cancer in particular. Way back in 1982, the National Research Council in the United States published a technical report entitled “Diet Nutrition and Cancer,” which showed that diet was probably the single most important factor in the development of cancer and that there was evidence linking cancer of the breast, colon, and prostate to particular foods or types of food. But the evidence linking cancer and food goes back many years before 1982. For example, since the mid-1970s there has been strong evidence to show a link between a high fat intake and a high risk for prostate cancer.
Studies in forty-one countries have shown a high correlation between mortality from prostate cancer and the intake of fats, milk, and meats (especially beef). In 1993 a study of 47,855 men reported in a medical research modernization committee report revealed that men who ate a high-fat diet had a relative risk of 1-79 for advanced prostate cancer as compared to those on a low-fat diet. Men eating a high-fat diet are almost twice as likely to develop prostate cancer as men on the low fat-diet are. Dr Ernst Wynder, director of the American Health Foundation called for more aggressive intervention studies in which men with early prostate cancer would be placed on a carefully controlled low-fat diet to see if it reduced their chance of dying of prostate cancer. Such a study was about to be done by the foundation, scrutinizing 2,000 women who had been treated for early- stage breast cancer. I have not seen the ﬁndings from this study.
Numerous studies have shown that a high intake of tomatoes markedly reduces the risk of prostate cancer. It is believed that this beneﬁcial eﬀect is due to lycopene, the most common carotenoid in tomatoes. A team of researchers from Wayne State University, McGill University, the University of Maryland, has concluded a clinical trial aimed at evaluating the beneﬁts of lycopene supplementation in prostate cancer patients. The study included twenty-six men with clinical localized prostate cancer who were scheduled to undergo radical prostatectomy.
The men randomized into a control group and an intervention group. The intervention group received one ﬁfteen mg lycopene capsule with breakfast and dinner for three weeks prior to surgery. Blood samples were taken before the start of the supplementation and three weeks prior to surgery. They removed the tumor, and surrounding tissue was examined by pathologists.
The researchers conclude that lycopene supplementation lowers PSA levels. They observed an average of an 18 percent decrease in the lycopene group as compared to a 14 percent increase in the control group. The level of the tumor-suppressing protein CX43 in the malignant part of the tumor was found to be substantially higher in the lycopene group. It was also apparent that tumors tended to be smaller and more sharply deﬁned (less encroachment into surrounding healthy tissue) in the lycopene group. No adverse eﬀects of lycopene supplementation were reported by the patients or the physicians. The researchers conclude that lycopene is likely to be beneﬁcial for both prevention and treatment of prostate cancer, but they urge larger trials to conﬁrm this.
In Staﬀordshire, in the UK, British researchers have conﬁrmed that exposure to sunlight helps prevent prostate cancer. Their study involved 210 men diagnosed with prostate cancer and 155 men with an enlarged prostate, but no prostate cancer (controls). The men were interviewed in order to estimate their lifetime sun exposure. Men with the lowest exposure were found to have a three-times greater incidence of prostate cancer than did men with high lifetime exposure. Sunburns in childhood were found to be particularly protective, with men having had one or more childhood sunburns being six times less likely to develop prostate cancer than did men who did not experience childhood sunburns. The researchers are not sure why sun exposure is protective, but speculate that vitamin D and the parathyroid hormone may somehow be involved.
Bible verses for today’s meditation and inspiration: Matthew E. McLaren
Rejoice with those who rejoice; mourn with those who mourn. Romans 12:15 NIV
You are my hiding place; you will protect me from trouble and surround me with songs of deliverance.
Psalm 32:7 NIV
So that I may come to you with joy, by God’s will, and in your company be refreshed. Romans 15:32 NIV
Your statutes are my heritage forever; they are the joy of my heart. Psalm 119:111 NIV
But godliness with contentment is great gain. 1 Timothy 6:6 NIV
And without faith it is impossible to please God, because anyone who comes to him must believe that he exists and that he rewards those who earnestly seek him. Hebrews 11:6 NIV
For the Lord takes delight in his people; he crowns the humble with victory. Psalm 149:4 NIV
Command those who are rich in this present world not to be arrogant nor to put their hope in wealth, which is so uncertain, but to put their hope in God, who richly provides us with everything for our enjoyment. 1 Timothy 6:17 NIV
The father of a righteous child has great joy; a man who fathers a wise son rejoices in him. Proverbs 23:24 NIV
I rejoice in following your statutes as one rejoices in great riches. Psalm 119:14 NIV
While he was still speaking, a bright cloud covered them, and a voice from the cloud said, “This is my Son, whom I love; with him I am well pleased. Listen to him!” Matthew 17:5 NIV
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